Contraction stress test

A contraction stress test (CST) is performed near the end of pregnancy (34 weeks' gestation) to determine how well the fetus will cope with the contractions of childbirth. The aim is to induce contractions and monitor the fetus to check for heart rate abnormalities using a cardiotocograph. A CST is one type of antenatal fetal surveillance technique.

Contraction stress test
Purposefetal test(heart rate)

During uterine contractions, fetal oxygenation is worsened. Late decelerations in fetal heart rate occurring during uterine contractions are associated with increased fetal death rate, growth retardation and neonatal depression.[1][2] This test assesses fetal heart rate in response to uterine contractions via electronic fetal monitoring. Uterine activity is monitored by tocodynamometer.[3]

Medical uses

The CST is used for its high negative predictive value. A negative result is highly predictive of fetal wellbeing and tolerance of labor. The test has a poor positive predictive value with false-positive results in as many as 30% of cases.[4][5] A positive CST indicates high risk of fetal death due to hypoxia[3] and is a contraindication to labor. Patient's obstetricians usually consider operative delivery in such situations.[6]

Contraindications

This "stress test" is usually not performed if there are any signs of premature birth, placenta praevia, vasa praevia, cervical incompetence, multiple gestation, previous classic caesarian section.[1] Other contraindications include but are not limited to previous uterine incision with scarring, previous myomectomy entering the uterine cavity, and PROM. Any contraindication to labor is contraindication to CST.

Procedure

CST is performed weekly,[1] as the fetus is assumed to be healthy after a negative test and should remain so for another week.[7] This test is done in hospital or clinic setting.[3] External fetal monitors are put in place and then either nipple stimulation or IV pitocin (oxytocin) is used to stimulate uterine contractions.

Nipple stimulation

This is a procedure that relies on endogenous release of oxytocin following nipple stimulation, and is conducted by the patient. The nurse instructs the patient on the procedure, as follows. One nipple is massaged gently through clothing until a contraction begins, or for a maximum of 2 minutes. If at least 3 contractions in 10 minutes is not achieved, then the patient rests for 5 minutes and the other nipple is stimulated.[8][4]

Oxytocin challenge test (OCT)

If adequate contractions (at least 3 in 10 minutes) cannot be achieved with nipple stimulation, an oxytocin challenge test may be performed. It involves the intravenous administration of exogenous oxytocin to the pregnant woman. The target is to achieve around three contractions every ten minutes.[8][4]

Interpretation
ResultInterpretation
PositivePresence of late decelerations with at least 50% of the contractions[9]
NegativeNo late or significant variable decelerations,[9] with at least 3 uterine contractions (lasting 40 seconds) in 10 minute period.[10]
Equivocal—SuspiciousPresence of late decelerations with fewer than 50% of contractions or significant variable decelerations. Requires repeat testing on following day.[1]
Equivocal—TachysystolePresence of contractions that occur more frequently than every 2 minutes or last longer than 90 seconds in the presence of late decelerations. Requires repeat testing on following day.[1]
Equivocal—UnsatisfactoryFewer than three contractions occur within 10 minutes, or a tracing quality that cannot be interpreted. Requires repeat testing on following day.[1]

History

The CST was the first antenatal surveillance test that was developed after the development of the cardiotocograph.[4] The oxytocin challenge test was first described in 1972[11] and was standardised in 1975 when the parameters of contraction number and frequency were given.

Historically, a CST was done after a non reactive NST. Today, a biophysical profile (BPP) is usually performed.

See also

References
  1. Ronald S. Gibbs; et al., eds. (2008). Danforth's obstetrics and gynecology (10th ed.). Philadelphia: Lippincott Williams & Wilkins. p. 161. ISBN 9780781769372.
  2. Alan H. DeCherney; T. Murphy Goodwin; et al., eds. (2007). Current diagnosis & treatment : Obstetrics & gynecology (10th ed.). New York: McGraw-Hill. pp. 255. ISBN 978-0-07-143900-8.
  3. III, Frances Talaska Fischbach, Marshall Barnett Dunning (2009). A manual of laboratory and diagnostic tests (8th ed.). Philadelphia: Wolters Kluwer Health/Lippincott Williams & Wilkins. pp. 1030–31. ISBN 9780781771948.{{cite book}}: CS1 maint: multiple names: authors list (link)
  4. Association of Women's Health, Obstetric, and Neonatal Nurses (2005). Audrey Lyndon; Linda Usher Ali (eds.). Fetal Heart Monitoring: Principles and Practices (3rd ed.). Dubuque, IA: Kendall/Hunt Publishing Co. ISBN 978-0-7575-6234-1.
  5. Lagrew DC Jr (March 1995). "The contraction stress test". Clinical Obstetrics and Gynecology. 38 (1): 11–25. doi:10.1097/00003081-199503000-00005. PMID 7796539. S2CID 45260930.
  6. Tao Le; et al. (2008). First aid for the family medicine boards. New York: McGraw-Hill Medical. pp. 556. ISBN 978-0-07-159382-3.
  7. Munden, ed.: Julie (2005). Professional guide to diagnostic tests. Philadelphia: Lippincott Williams & Wilkins. pp. 682. ISBN 9781582553047. {{cite book}}: |first= has generic name (help)
  8. American College of Obstetricians and Gynecologists (ACOG). (1999). Antepartum fetal surveillance (Practice Bulletin No. 9). Washington, DC: Author.
  9. Evans (2007). Arthur T. (ed.). Manual of obstetrics (7th ed.). Philadelphia: Wolters Kluwer / Lippincott Williams & Wilkins. p. 587. ISBN 9780781796965.
  10. Anderson (2005). Jean R. (ed.). A guide to the clinical care of women with HIV (2005 ed.). Rockville, MD: U.S. Dept. of Health & Human Services, Health Resources & Services Administration, HIV/AIDS Bureau. p. 270. ISBN 9780160726118.
  11. Ray M, Freeman R, Pine S, Hesselgesser R (September 1972). "Clinical experience with the oxytocin challenge test". Am. J. Obstet. Gynecol. 114 (1): 1–9. doi:10.1016/0002-9378(72)90279-7. PMID 4637035.
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