GTF3C2
GTF3C2 (General transcription factor IIIC subunit 2) هوَ بروتين يُشَفر بواسطة جين GTF3C2 في الإنسان.[1][2]
المراجع
- "Cloning and characterization of a TFIIIC2 subunit (TFIIIC beta) whose presence correlates with activation of RNA polymerase III-mediated transcription by adenovirus E1A expression and serum factors"، Genes Dev.، 9 (6): 675–85، يونيو 1995، doi:10.1101/gad.9.6.675، PMID 7729686.
- "Entrez Gene: GTF3C2 general transcription factor IIIC, polypeptide 2, beta 110kDa"، مؤرشف من الأصل في 05 ديسمبر 2010.
قراءة متعمقة
- "The human immunodeficiency virus tat protein increases the transcription of human Alu repeated sequences by increasing the activity of the cellular transcription factor TFIIIC"، J. Acquir. Immune Defic. Syndr.، 5 (11): 1142–7، 1992، PMID 1403646.
- "Prediction of the coding sequences of unidentified human genes. I. The coding sequences of 40 new genes (KIAA0001-KIAA0040) deduced by analysis of randomly sampled cDNA clones from human immature myeloid cell line KG-1"، DNA Res.، 1 (1): 27–35، 1994، doi:10.1093/dnares/1.1.27، PMID 7584026.
- "Prediction of the coding sequences of unidentified human genes. I. The coding sequences of 40 new genes (KIAA0001-KIAA0040) deduced by analysis of randomly sampled cDNA clones from human immature myeloid cell line KG-1 (supplement)"، DNA Res.، 1 (1): 47–56، 1994، doi:10.1093/dnares/1.1.47، PMID 7584028.
- "A "double adaptor" method for improved shotgun library construction"، Anal. Biochem.، 236 (1): 107–13، 1996، doi:10.1006/abio.1996.0138، PMID 8619474.
- "Large-scale concatenation cDNA sequencing"، Genome Res.، 7 (4): 353–8، 1997، doi:10.1101/gr.7.4.353، PMC 139146، PMID 9110174.
- "RNA polymerase III transcription repressed by Rb through its interactions with TFIIIB and TFIIIC2"، J. Biol. Chem.، 272 (23): 14755–61، 1997، doi:10.1074/jbc.272.23.14755، PMID 9169441.
- "Human transcription factors IIIC2 , IIIC1 and a novel component IIIC0 fulfil different aspects of DNA binding to various pol III genes"، Nucleic Acids Res.، 25 (12): 2440–7، 1997، doi:10.1093/nar/25.12.2440، PMC 146769، PMID 9171097.
- "DNA topoisomerase I and PC4 can interact with human TFIIIC to promote both accurate termination and transcription reinitiation by RNA polymerase III"، Mol. Cell، 1 (5): 749–57، 1998، doi:10.1016/S1097-2765(00)80074-X، PMID 9660958.
- "Cloning and characterization of two evolutionarily conserved subunits (TFIIIC102 and TFIIIC63) of human TFIIIC and their involvement in functional interactions with TFIIIB and RNA polymerase III"، Mol. Cell. Biol.، 19 (7): 4944–52، 1999، PMC 84305، PMID 10373544.
- "The TFIIIC90 subunit of TFIIIC interacts with multiple components of the RNA polymerase III machinery and contains a histone-specific acetyltransferase activity"، Mol. Cell. Biol.، 19 (11): 7697–704، 1999، doi:10.1128/mcb.19.11.7697، PMC 84812، PMID 10523658.
- "Sequence comparison of human and mouse genes reveals a homologous block structure in the promoter regions"، Genome Res.، 14 (9): 1711–8، 2004، doi:10.1101/gr.2435604، PMC 515316، PMID 15342556.
- "Towards a proteome-scale map of the human protein-protein interaction network"، Nature، 437 (7062): 1173–8، 2005، doi:10.1038/nature04209، PMID 16189514.
- "A test of the model that RNA polymerase III transcription is regulated by selective induction of the 110 kDa subunit of TFIIIC"، Nucleic Acids Res.، 34 (11): 3399–407، 2006، doi:10.1093/nar/gkl432، PMC 1488882، PMID 16822860.
- "A probability-based approach for high-throughput protein phosphorylation analysis and site localization"، Nat. Biotechnol.، 24 (10): 1285–92، 2006، doi:10.1038/nbt1240، PMID 16964243.
- "Global, in vivo, and site-specific phosphorylation dynamics in signaling networks"، Cell، 127 (3): 635–48، 2006، doi:10.1016/j.cell.2006.09.026، PMID 17081983.
- "Identification, molecular cloning, and characterization of the sixth subunit of human transcription factor TFIIIC"، J. Biol. Chem.، 282 (23): 17179–89، 2007، doi:10.1074/jbc.M611542200، PMID 17409385.
بوابة علم الأحياء الخلوي والجزيئي
بوابة طب
بوابة الكيمياء الحيوية
This article is issued from Wikipedia. The text is licensed under Creative Commons - Attribution - Sharealike. Additional terms may apply for the media files.