BCAR3
BCAR3 (BCAR3, NSP family adaptor protein) هوَ بروتين يُشَفر بواسطة جين BCAR3 في الإنسان.[1][2]
المراجع
- "Identification of BCAR3 by a random search for genes involved in antiestrogen resistance of human breast cancer cells"، EMBO J، 17 (10): 2799–808، يوليو 1998، doi:10.1093/emboj/17.10.2799، PMC 1170620، PMID 9582273.
- "Entrez Gene: BCAR3 breast cancer anti-estrogen resistance 3"، مؤرشف من الأصل في 05 ديسمبر 2010.
قراءة متعمقة
- "Genetic mechanisms of estrogen-independence in breast cancer."، Pathol. Res. Pract.، 192 (7): 743–51، 1997، doi:10.1016/S0344-0338(96)80096-3، PMID 8880875.
- Johnston SR (1998)، "Acquired tamoxifen resistance in human breast cancer--potential mechanisms and clinical implications."، Anticancer Drugs، 8 (10): 911–30، doi:10.1097/00001813-199711000-00002، PMID 9436634.
- "NSP1 defines a novel family of adaptor proteins linking integrin and tyrosine kinase receptors to the c-Jun N-terminal kinase/stress-activated protein kinase signaling pathway."، J. Biol. Chem.، 274 (15): 10047–52، 1999، doi:10.1074/jbc.274.15.10047، PMID 10187783.
- "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences."، Proc. Natl. Acad. Sci. U.S.A.، 99 (26): 16899–903، 2003، doi:10.1073/pnas.242603899، PMC 139241، PMID 12477932.
- "The GDP exchange factor AND-34 is expressed in B cells, associates with HEF1, and activates Cdc42."، J. Immunol.، 170 (2): 969–78، 2003، doi:10.4049/jimmunol.170.2.969، PMID 12517963.
- "The secreted protein discovery initiative (SPDI), a large-scale effort to identify novel human secreted and transmembrane proteins: a bioinformatics assessment."، Genome Res.، 13 (10): 2265–70، 2003، doi:10.1101/gr.1293003، PMC 403697، PMID 12975309.
- "Complete sequencing and characterization of 21,243 full-length human cDNAs."، Nat. Genet.، 36 (1): 40–5، 2004، doi:10.1038/ng1285، PMID 14702039.
- "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)."، Genome Res.، 14 (10B): 2121–7، 2004، doi:10.1101/gr.2596504، PMC 528928، PMID 15489334.
- "Breast cancer oestrogen independence mediated by BCAR1 or BCAR3 genes is transmitted through mechanisms distinct from the oestrogen receptor signalling pathway or the epidermal growth factor receptor signalling pathway."، Breast Cancer Res.، 7 (1): R82–92، 2006، doi:10.1186/bcr954، PMC 1064102، PMID 15642172.
- "AND-34 activates phosphatidylinositol 3-kinase and induces anti-estrogen resistance in a SH2 and GDP exchange factor-like domain-dependent manner."، Mol. Cancer Res.، 3 (1): 32–41، 2005، PMID 15671247.
- "The DNA sequence and biological annotation of human chromosome 1."، Nature، 441 (7091): 315–21، 2006، doi:10.1038/nature04727، PMID 16710414.
- "A probability-based approach for high-throughput protein phosphorylation analysis and site localization."، Nat. Biotechnol.، 24 (10): 1285–92، 2006، doi:10.1038/nbt1240، PMID 16964243.
- "Global, in vivo, and site-specific phosphorylation dynamics in signaling networks."، Cell، 127 (3): 635–48، 2006، doi:10.1016/j.cell.2006.09.026، PMID 17081983.
- "Splice variants and expression patterns of SHEP1, BCAR3 and NSP1, a gene family involved in integrin and receptor tyrosine kinase signaling."، Gene، 391 (1–2): 161–70، 2007، doi:10.1016/j.gene.2006.12.016، PMC 1876674، PMID 17270363.
- "Large-scale mapping of human protein-protein interactions by mass spectrometry."، Mol. Syst. Biol.، 3 (1): 89، 2007، doi:10.1038/msb4100134، PMC 1847948، PMID 17353931.
- "AND-34/BCAR3 differs from other NSP homologs in induction of anti-estrogen resistance, cyclin D1 promoter activation and altered breast cancer cell morphology."، J. Cell. Physiol.، 212 (3): 655–65، 2007، doi:10.1002/jcp.21059، PMC 2640322، PMID 17427198.
- "Breast cancer antiestrogen resistance-3 expression regulates breast cancer cell migration through promotion of p130Cas membrane localization and membrane ruffling."، Cancer Res.، 67 (13): 6174–82، 2007، doi:10.1158/0008-5472.CAN-06-3455، PMC 4109708، PMID 17616674.
بوابة الكيمياء الحيوية
بوابة طب
بوابة علم الأحياء الخلوي والجزيئي
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