NFKBIZ
NFKBIZ (NFKB inhibitor zeta) هوَ بروتين يُشَفر بواسطة جين NFKBIZ في الإنسان.[1][2][3]
الوظيفة
المراجع
- "Entrez Gene: NFKBIZ nuclear factor of kappa light polypeptide gene enhancer in B-cells inhibitor, zeta"، مؤرشف من الأصل في 05 ديسمبر 2010.
- "A novel member of the IkappaB family, human IkappaB-zeta, inhibits transactivation of p65 and its DNA binding"، J Biol Chem، 281 (18): 12645–54، مايو 2006، doi:10.1074/jbc.M511956200، PMID 16513645.
- "Essential roles for NF-kappa B and a Toll/IL-1 receptor domain-specific signal(s) in the induction of I kappa B-zeta"، Biochem Biophys Res Commun، 301 (2): 495–501، فبراير 2003، doi:10.1016/S0006-291X(02)03082-6، PMID 12565889.
قراءة متعمقة
- "MAIL, a novel nuclear I kappa B protein that potentiates LPS-induced IL-6 production."، FEBS Lett.، 485 (1): 53–6، 2000، doi:10.1016/S0014-5793(00)02185-2، PMID 11086164.
- "Genomic organization, chromosomal localization, and promoter analysis of the mouse Mail gene."، Immunogenetics، 53 (8): 649–55، 2001، doi:10.1007/s00251-001-0376-x، PMID 11797098.
- "Isolation of a novel interleukin-1-inducible nuclear protein bearing ankyrin-repeat motifs."، J. Biol. Chem.، 276 (16): 12485–8، 2001، doi:10.1074/jbc.C100075200، PMID 11278262.
- "A novel IkappaB protein, IkappaB-zeta, induced by proinflammatory stimuli, negatively regulates nuclear factor-kappaB in the nuclei."، J. Biol. Chem.، 276 (29): 27657–62، 2001، doi:10.1074/jbc.M103426200، PMID 11356851.
- "Bacterial lipopolysaccharide induces mRNA expression of an IkappaB MAIL through toll-like receptor 4."، J. Vet. Med. Sci.، 64 (5): 419–22، 2002، doi:10.1292/jvms.64.419، PMID 12069074.
- "Bacterial lipopolysaccharide-induced expression of the IkappaB protein MAIL in B-lymphocytes and macrophages."، Arch. Histol. Cytol.، 66 (1): 53–62، 2003، doi:10.1679/aohc.66.53، PMID 12703554.
- "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences."، Proc. Natl. Acad. Sci. U.S.A.، 99 (26): 16899–903، 2003، doi:10.1073/pnas.242603899، PMC 139241، PMID 12477932.
- "Targeted disruption of MAIL, a nuclear IkappaB protein, leads to severe atopic dermatitis-like disease."، J. Biol. Chem.، 279 (53): 55493–8، 2004، doi:10.1074/jbc.M409770200، PMID 15491998.
- "Transcriptional regulation of the MAIL gene in LPS-stimulated RAW264 mouse macrophages."، Gene، 342 (1): 137–43، 2004، doi:10.1016/j.gene.2004.07.032، PMID 15527973.
- "Complete sequencing and characterization of 21,243 full-length human cDNAs."، Nat. Genet.، 36 (1): 40–5، 2004، doi:10.1038/ng1285، PMID 14702039.
- "Regulation of Toll/IL-1-receptor-mediated gene expression by the inducible nuclear protein IkappaBzeta."، Nature، 430 (6996): 218–22، 2004، doi:10.1038/nature02738، PMID 15241416.
- "Cloning of bovine MAIL and its mRNA expression in white blood cells of Holstein cows."، Vet. Immunol. Immunopathol.، 98 (3–4): 175–84، 2004، doi:10.1016/j.vetimm.2003.12.004، PMID 15010226.
- "Stimulus-specific induction of a novel nuclear factor-kappaB regulator, IkappaB-zeta, via Toll/Interleukin-1 receptor is mediated by mRNA stabilization."، J. Biol. Chem.، 280 (2): 1678–87، 2005، doi:10.1074/jbc.M409983200، PMID 15522867.
- "Positive and negative regulation of nuclear factor-kappaB-mediated transcription by IkappaB-zeta, an inducible nuclear protein."، J. Biol. Chem.، 280 (9): 7444–51، 2005، doi:10.1074/jbc.M412738200، PMID 15618216.
- "IL-1beta-specific up-regulation of neutrophil gelatinase-associated lipocalin is controlled by IkappaB-zeta."، J. Immunol.، 176 (9): 5559–66، 2006، doi:10.4049/jimmunol.176.9.5559، PMID 16622025.
- "Role of NF-kappaB in constitutive expression of MAIL in epidermal keratinocytes."، J Vet Med Sci.، 69 (3): 279–84، 2007، doi:10.1292/jvms.69.279، PMID 17409644.
- "Crucial roles of binding sites for NF-kappaB and C/EBPs in IkappaB-zeta-mediated transcriptional activation."، Biochem. J.، 405 (3): 605–15، 2007، doi:10.1042/BJ20061797، PMC 2267307، PMID 17447895.
بوابة طب
بوابة الكيمياء الحيوية
بوابة علم الأحياء الخلوي والجزيئي
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