CPLX2
CPLX2 (Complexin 2) هوَ بروتين يُشَفر بواسطة جين CPLX2 في الإنسان.[1][2][3]
المراجع
- "Structural organization of the human complexin 2 gene (CPLX2) and aspects of its functional activity"، Gene، 359: 127–37، سبتمبر 2005، doi:10.1016/j.gene.2005.07.005، PMID 16162394.
- "Entrez Gene: CPLX2 complexin 2"، مؤرشف من الأصل في 05 ديسمبر 2010.
- "Complexins: cytosolic proteins that regulate SNAP receptor function"، Cell، 83 (1): 111–9، نوفمبر 1995، doi:10.1016/0092-8674(95)90239-2، PMID 7553862.
قراءة متعمقة
- Abe T (2002)، "[The mechanism of neurotransmitter release: role of synaphin/complexin in synaptic vesicle exocytosis]"، Tanpakushitsu Kakusan Koso، 47 (7): 794–800، PMID 12058476.
- "Effect of time of day of insemination and the position of the egg in the oviduct on the fertility of turkeys"، Poult. Sci.، 56 (2): 458–62، 1978، doi:10.3382/ps.0560458، PMID 605033.
- Morgans CW؛ Brandstätter JH؛ Kellerman J؛ وآخرون (1996)، "A SNARE complex containing syntaxin 3 is present in ribbon synapses of the retina"، J. Neurosci.، 16 (21): 6713–21، PMID 8824312.
- "Preferential involvement of excitatory neurons in medial temporal lobe in schizophrenia"، Lancet، 352 (9141): 1669–73، 1998، doi:10.1016/S0140-6736(98)03341-8، PMID 9853440.
- Ishizuka T؛ Saisu H؛ Odani S؛ وآخرون (1999)، "Distinct regional distribution in the brain of messenger RNAs for the two isoforms of synaphin associated with the docking/fusion complex"، Neuroscience، 88 (1): 295–306، doi:10.1016/S0306-4522(98)00223-1، PMID 10051208.
- Yamada M؛ Saisu H؛ Ishizuka T؛ وآخرون (1999)، "Immunohistochemical distribution of the two isoforms of synaphin/complexin involved in neurotransmitter release: localization at the distinct central nervous system regions and synaptic types"، Neuroscience، 93 (1): 7–18، doi:10.1016/S0306-4522(99)00104-9، PMID 10430466.
- Pabst S؛ Hazzard JW؛ Antonin W؛ وآخرون (2000)، "Selective interaction of complexin with the neuronal SNARE complex. Determination of the binding regions"، J. Biol. Chem.، 275 (26): 19808–18، doi:10.1074/jbc.M002571200، PMID 10777504.
- "Cerebellar synaptic protein expression in schizophrenia"، Neuroscience، 105 (1): 219–29، 2001، doi:10.1016/S0306-4522(01)00141-5، PMID 11483314.
- "Synaptic pathology in the anterior cingulate cortex in schizophrenia and mood disorders. A review and a Western blot study of synaptophysin, GAP-43 and the complexins"، Brain Res. Bull.، 55 (5): 569–78، 2001، doi:10.1016/S0361-9230(01)00530-5، PMID 11576753.
- Pabst S؛ Margittai M؛ Vainius D؛ وآخرون (2002)، "Rapid and selective binding to the synaptic SNARE complex suggests a modulatory role of complexins in neuroexocytosis"، J. Biol. Chem.، 277 (10): 7838–48، doi:10.1074/jbc.M109507200، PMID 11751907.
- Sawada K؛ Young CE؛ Barr AM؛ وآخرون (2002)، "Altered immunoreactivity of complexin protein in prefrontal cortex in severe mental illness"، Mol. Psychiatry، 7 (5): 484–92، doi:10.1038/sj.mp.4000978، PMID 12082566.
- "Action of complexin on SNARE complex"، J. Biol. Chem.، 277 (44): 41652–6، 2003، doi:10.1074/jbc.M205044200، PMID 12200427.
- Strausberg RL؛ Feingold EA؛ Grouse LH؛ وآخرون (2003)، "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences"، Proc. Natl. Acad. Sci. U.S.A.، 99 (26): 16899–903، doi:10.1073/pnas.242603899، PMC 139241، PMID 12477932.
- Edwardson JM؛ Wang CT؛ Gong B؛ وآخرون (2003)، "Expression of mutant huntingtin blocks exocytosis in PC12 cells by depletion of complexin II"، J. Biol. Chem.، 278 (33): 30849–53، doi:10.1074/jbc.M304615200، PMID 12807877.
- Ota T؛ Suzuki Y؛ Nishikawa T؛ وآخرون (2004)، "Complete sequencing and characterization of 21,243 full-length human cDNAs"، Nat. Genet.، 36 (1): 40–5، doi:10.1038/ng1285، PMID 14702039.
- Gerhard DS؛ Wagner L؛ Feingold EA؛ وآخرون (2004)، "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)"، Genome Res.، 14 (10B): 2121–7، doi:10.1101/gr.2596504، PMC 528928، PMID 15489334.
- "Decreased expression of vesicular glutamate transporter 1 and complexin II mRNAs in schizophrenia: further evidence for a synaptic pathology affecting glutamate neurons"، Schizophr. Res.، 73 (2–3): 159–72، 2005، doi:10.1016/j.schres.2004.05.010، PMID 15653259.
- DiProspero NA؛ Chen EY؛ Charles V؛ وآخرون (2005)، "Early changes in Huntington's disease patient brains involve alterations in cytoskeletal and synaptic elements"، J. Neurocytol.، 33 (5): 517–33، doi:10.1007/s11068-004-0514-8، PMID 15906159.
بوابة الكيمياء الحيوية
بوابة علم الأحياء الخلوي والجزيئي
بوابة طب
This article is issued from Wikipedia. The text is licensed under Creative Commons - Attribution - Sharealike. Additional terms may apply for the media files.