BCL11B
BCL11B (B cell CLL/lymphoma 11B) هوَ بروتين يُشَفر بواسطة جين BCL11B في الإنسان.[1][2][3]
المراجع
- "Entrez Gene: BCL11B B-cell CLL/lymphoma 11B (zinc finger protein)"، مؤرشف من الأصل في 05 ديسمبر 2010.
- "CTIP2 associates with the NuRD complex on the promoter of p57KIP2, a newly identified CTIP2 target gene"، J. Biol. Chem.، 281 (43): 32272–83، أكتوبر 2006، doi:10.1074/jbc.M602776200، PMC 2547407، PMID 16950772.
- "The BCL11 gene family: involvement of BCL11A in lymphoid malignancies"، Blood، 98 (12): 3413–20، نوفمبر 2001، doi:10.1182/blood.V98.12.3413، PMID 11719382.
قراءة متعمقة
- "Isolation of a novel family of C(2)H(2) zinc finger proteins implicated in transcriptional repression mediated by chicken ovalbumin upstream promoter transcription factor (COUP-TF) orphan nuclear receptors"، J. Biol. Chem.، 275 (14): 10315–22، 2000، doi:10.1074/jbc.275.14.10315، PMC 2819356، PMID 10744719.
- "COUP-TF (chicken ovalbumin upstream promoter transcription factor)-interacting protein 1 (CTIP1) is a sequence-specific DNA binding protein"، Biochem. J.، 368 (Pt 2): 555–63، 2002، doi:10.1042/BJ20020496، PMC 1223006، PMID 12196208.
- "Homozygous deletions and point mutations of the Rit1/Bcl11b gene in gamma-ray induced mouse thymic lymphomas"، Biochem. Biophys. Res. Commun.، 301 (2): 598–603، 2003، doi:10.1016/S0006-291X(02)03069-3، PMID 12565905.
- "Recruitment of Tat to heterochromatin protein HP1 via interaction with CTIP2 inhibits human immunodeficiency virus type 1 replication in microglial cells"، J. Virol.، 77 (9): 5415–27، 2003، doi:10.1128/JVI.77.9.5415-5427.2003، PMC 153947، PMID 12692243.
- "Involvement of the histone deacetylase SIRT1 in chicken ovalbumin upstream promoter transcription factor (COUP-TF)-interacting protein 2-mediated transcriptional repression"، J. Biol. Chem.، 278 (44): 43041–50، 2003، doi:10.1074/jbc.M307477200، PMC 2819354، PMID 12930829.
- "A novel t(6;14)(q25-q27;q32) in acute myelocytic leukemia involves the BCL11B gene"، Cancer Genet. Cytogenet.، 149 (1): 72–6، 2004، doi:10.1016/S0165-4608(03)00302-9، PMID 15104287.
- "BCL11B rearrangements probably target T-cell neoplasia rather than acute myelocytic leukemia"، Cancer Genet. Cytogenet.، 153 (1): 88–9، 2004، doi:10.1016/j.cancergencyto.2004.02.020، PMID 15325104.
- "Disruption of the BCL11B gene through inv(14)(q11.2q32.31) results in the expression of BCL11B-TRDC fusion transcripts and is associated with the absence of wild-type BCL11B transcripts in T-ALL"، Leukemia، 19 (2): 201–8، 2005، doi:10.1038/sj.leu.2403619، PMID 15668700.
- "BCL11B functionally associates with the NuRD complex in T lymphocytes to repress targeted promoter"، Oncogene، 24 (45): 6753–64، 2005، doi:10.1038/sj.onc.1208904، PMID 16091750.
- "Recruitment of chromatin-modifying enzymes by CTIP2 promotes HIV-1 transcriptional silencing"، EMBO J.، 26 (2): 412–23، 2007، doi:10.1038/sj.emboj.7601516، PMC 1783449، PMID 17245431.
- "Activation of TLX3 and NKX2-5 in t(5;14)(q35;q32) T-cell acute lymphoblastic leukemia by remote 3'-BCL11B enhancers and coregulation by PU.1 and HMGA1"، Cancer Res.، 67 (4): 1461–71، 2007، doi:10.1158/0008-5472.CAN-06-2615، PMID 17308084.
- "Lack of Bcl11b tumor suppressor results in vulnerability to DNA replication stress and damages"، Oncogene، 26 (40): 5840–50، 2007، doi:10.1038/sj.onc.1210388، PMID 17369851.
- بوابة علم الأحياء الخلوي والجزيئي
- بوابة الكيمياء الحيوية
- بوابة طب
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