BCL2L2
BCL2L2 (BCL2 like 2) هوَ بروتين يُشَفر بواسطة جين BCL2L2 في الإنسان.[1][2][3]
الوظيفة
المراجع
- "bcl-w, a novel member of the bcl-2 family, promotes cell survival"، Oncogene، 13 (4): 665–75، أكتوبر 1996، PMID 8761287.
- "bcl-w, a novel member of the bcl-2 family, promotes cell survival."، Oncogene، 13 (4): 665–75، 1996، PMID 8761287.
- "Entrez Gene: BCL2L2 BCL2-like 2"، مؤرشف من الأصل في 05 ديسمبر 2010.
قراءة متعمقة
- "Prediction of the coding sequences of unidentified human genes. VI. The coding sequences of 80 new genes (KIAA0201-KIAA0280) deduced by analysis of cDNA clones from cell line KG-1 and brain."، DNA Res.، 3 (5): 321–9, 341–54، 1997، doi:10.1093/dnares/3.5.321، PMID 9039502.
- "Bim: a novel member of the Bcl-2 family that promotes apoptosis."، EMBO J.، 17 (2): 384–95، 1998، doi:10.1093/emboj/17.2.384، PMC 1170389، PMID 9430630.
- "Testicular degeneration in Bclw-deficient mice."، Nat. Genet.، 18 (3): 251–6، 1998، doi:10.1038/ng0398-251، PMID 9500547.
- "BOD (Bcl-2-related ovarian death gene) is an ovarian BH3 domain-containing proapoptotic Bcl-2 protein capable of dimerization with diverse antiapoptotic Bcl-2 members."، Mol. Endocrinol.، 12 (9): 1432–40، 1998، doi:10.1210/mend.12.9.0166، PMID 9731710.
- "Reciprocal developmental changes in the roles of Bcl-w and Bcl-x(L) in regulating sensory neuron survival."، Development، 128 (3): 447–57، 2001، PMID 11152643.
- "Tissue expression and subcellular localization of the pro-survival molecule Bcl-w."، Cell Death Differ.، 8 (5): 486–94، 2001، doi:10.1038/sj.cdd.4400835، PMID 11423909.
- "Underphosphorylated BAD interacts with diverse antiapoptotic Bcl-2 family proteins to regulate apoptosis."، Apoptosis، 6 (5): 319–30، 2001، doi:10.1023/A:1011319901057، PMID 11483855.
- "Bmf: a proapoptotic BH3-only protein regulated by interaction with the myosin V actin motor complex, activated by anoikis."، Science، 293 (5536): 1829–32، 2001، doi:10.1126/science.1062257، PMID 11546872.
- "The anti-apoptotic molecules Bcl-xL and Bcl-w target protein phosphatase 1alpha to Bad."، Eur. J. Immunol.، 32 (7): 1847–55، 2002، doi:10.1002/1521-4141(200207)32:7<1847::AID-IMMU1847>3.0.CO;2-7، PMID 12115603.
- "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences."، Proc. Natl. Acad. Sci. U.S.A.، 99 (26): 16899–903، 2003، doi:10.1073/pnas.242603899، PMC 139241، PMID 12477932.
- "Solution structure of human BCL-w: modulation of ligand binding by the C-terminal helix."، J. Biol. Chem.، 278 (23): 21124–8، 2003، doi:10.1074/jbc.M301798200، PMID 12651847.
- "The structure of Bcl-w reveals a role for the C-terminal residues in modulating biological activity."، EMBO J.، 22 (7): 1497–507، 2003، doi:10.1093/emboj/cdg144، PMC 152889، PMID 12660157.
- "Proapoptotic BH3-only proteins trigger membrane integration of prosurvival Bcl-w and neutralize its activity."، J. Cell Biol.، 162 (5): 877–87، 2003، doi:10.1083/jcb.200302144، PMC 2172834، PMID 12952938.
- "Neuroprotective properties of Bcl-w in Alzheimer disease."، J. Neurochem.، 89 (5): 1233–40، 2004، doi:10.1111/j.1471-4159.2004.02416.x، PMID 15147516.
- "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)."، Genome Res.، 14 (10B): 2121–7، 2004، doi:10.1101/gr.2596504، PMC 528928، PMID 15489334.
- "Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function."، Mol. Cell، 17 (3): 393–403، 2005، doi:10.1016/j.molcel.2004.12.030، PMID 15694340.
- "Diversification of transcriptional modulation: large-scale identification and characterization of putative alternative promoters of human genes."، Genome Res.، 16 (1): 55–65، 2006، doi:10.1101/gr.4039406، PMC 1356129، PMID 16344560.
- "Structural model of the BCL-w-BID peptide complex and its interactions with phospholipid micelles."، Biochemistry، 45 (7): 2250–6، 2006، doi:10.1021/bi052332s، PMID 16475813.
- "Mitochondria primed by death signals determine cellular addiction to antiapoptotic BCL-2 family members."، Cancer Cell، 9 (5): 351–65، 2006، doi:10.1016/j.ccr.2006.03.027، PMID 16697956.
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بوابة الكيمياء الحيوية
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