SH3GL1
SH3GL1 (SH3 domain GRB2-like 1) هوَ بروتين يُشَفر بواسطة جين SH3GL1 في الإنسان.[1][2]
المراجع
- "Entrez Gene: SH3GL1 SH3-domain GRB2-like 1"، مؤرشف من الأصل في 05 ديسمبر 2010.
- "A novel SH3-containing human gene family preferentially expressed in the central nervous system"، Genomics، 41 (3): 427–34، يوليو 1997، doi:10.1006/geno.1997.4645، PMID 9169142.
قراءة متعمقة
- "EEN encodes for a member of a new family of proteins containing an Src homology 3 domain and is the third gene located on chromosome 19p13 that fuses to MLL in human leukemia."، Proc. Natl. Acad. Sci. U.S.A.، 94 (6): 2563–8، 1997، doi:10.1073/pnas.94.6.2563، PMC 20128، PMID 9122235.
- "The SH3p4/Sh3p8/SH3p13 protein family: binding partners for synaptojanin and dynamin via a Grb2-like Src homology 3 domain."، Proc. Natl. Acad. Sci. U.S.A.، 94 (16): 8569–74، 1997، doi:10.1073/pnas.94.16.8569، PMC 23017، PMID 9238017.
- "The SH3 domains of endophilin and amphiphysin bind to the proline-rich region of synaptojanin 1 at distinct sites that display an unconventional binding specificity."، J. Biol. Chem.، 274 (45): 32001–7، 1999، doi:10.1074/jbc.274.45.32001، PMID 10542231.
- "The interaction between EEN and Abi-1, two MLL fusion partners, and synaptojanin and dynamin: implications for leukaemogenesis."، Leukemia، 14 (4): 594–601، 2000، doi:10.1038/sj.leu.2401692، PMID 10764144.
- "Expression and protein-binding studies of the EEN gene family, new interacting partners for dynamin, synaptojanin and huntingtin proteins."، Biochem. J.، 348 (2): 447–58، 2000، doi:10.1042/0264-6021:3480447، PMC 1221085، PMID 10816441.
- "The endophilin-CIN85-Cbl complex mediates ligand-dependent downregulation of c-Met"، Nature، 416 (6877): 187–90، 2002، doi:10.1038/416187a، PMID 11894096.
- "Alix (ALG-2-interacting protein X), a protein involved in apoptosis, binds to endophilins and induces cytoplasmic vacuolization"، J. Biol. Chem.، 277 (32): 29108–15، 2002، doi:10.1074/jbc.M204019200، PMID 12034747.
- "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences"، Proc. Natl. Acad. Sci. U.S.A.، 99 (26): 16899–903، 2003، doi:10.1073/pnas.242603899، PMC 139241، PMID 12477932.
- "Identification and characterization of EBP, a novel EEN binding protein that inhibits Ras signaling and is recruited into the nucleus by the MLL-EEN fusion protein"، Blood، 103 (4): 1445–53، 2004، doi:10.1182/blood-2003-07-2452، PMID 14551139.
- "Complete sequencing and characterization of 21,243 full-length human cDNAs"، Nat. Genet.، 36 (1): 40–5، 2004، doi:10.1038/ng1285، PMID 14702039.
- "Functional contribution of EEN to leukemogenic transformation by MLL-EEN fusion protein"، Oncogene، 23 (19): 3385–94، 2004، doi:10.1038/sj.onc.1207402، PMID 15077184.
- "Subcellular localization of EEN/endophilin A2, a fusion partner gene in leukaemia"، Biochem. J.، 383 (Pt 1): 27–35، 2005، doi:10.1042/BJ20040041، PMC 1134040، PMID 15214844.
- "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)"، Genome Res.، 14 (10B): 2121–7، 2004، doi:10.1101/gr.2596504، PMC 528928، PMID 15489334.
- "Identification of novel CBP interacting proteins in embryonic orofacial tissue"، Biochem. Biophys. Res. Commun.، 329 (3): 1010–7، 2005، doi:10.1016/j.bbrc.2005.02.075، PMID 15752756.
- "Activation of EGF receptor endocytosis and ERK1/2 signaling by BPGAP1 requires direct interaction with EEN/endophilin II and a functional RhoGAP domain"، J. Cell Sci.، 118 (Pt 12): 2707–21، 2005، doi:10.1242/jcs.02383، PMID 15944398.
- "Ataxin-2 and huntingtin interact with endophilin-A complexes to function in plastin-associated pathways"، Hum. Mol. Genet.، 14 (19): 2893–909، 2005، doi:10.1093/hmg/ddi321، PMID 16115810.
- "A human protein-protein interaction network: a resource for annotating the proteome"، Cell، 122 (6): 957–68، 2005، doi:10.1016/j.cell.2005.08.029، PMID 16169070.
- "Towards a proteome-scale map of the human protein-protein interaction network"، Nature، 437 (7062): 1173–8، 2005، doi:10.1038/nature04209، PMID 16189514.
بوابة علم الأحياء الخلوي والجزيئي
بوابة طب
بوابة الكيمياء الحيوية
This article is issued from Wikipedia. The text is licensed under Creative Commons - Attribution - Sharealike. Additional terms may apply for the media files.